Koc et al., 2011 - Google Patents
Toll-like receptor expression in monocytes in patients with chronic kidney disease and haemodialysis: relation with inflammationKoc et al., 2011
View HTML- Document ID
- 8530124948776586645
- Author
- Koc M
- Toprak A
- Arikan H
- Odabasi Z
- Elbir Y
- Tulunay A
- Asicioglu E
- Eksioglu-Demiralp E
- Glorieux G
- Vanholder R
- Akoglu E
- Publication year
- Publication venue
- Nephrology Dialysis Transplantation
External Links
Snippet
Background. Inflammation is one of the main contributors to atherosclerosis in haemodialysis (HD) patients. Activation of Toll-like receptors (TLRs) leads to inflammatory response. In this study, we aimed to evaluate the expression of TLRs on monocytes and …
- 238000001631 haemodialysis 0 title abstract description 233
Classifications
-
- G—PHYSICS
- G01—MEASURING; TESTING
- G01N—INVESTIGATING OR ANALYSING MATERIALS BY DETERMINING THEIR CHEMICAL OR PHYSICAL PROPERTIES
- G01N33/00—Investigating or analysing materials by specific methods not covered by the preceding groups
- G01N33/48—Investigating or analysing materials by specific methods not covered by the preceding groups biological material, e.g. blood, urine; Haemocytometers
- G01N33/50—Chemical analysis of biological material, e.g. blood, urine; Testing involving biospecific ligand binding methods; Immunological testing
- G01N33/53—Immunoassay; Biospecific binding assay
- G01N33/569—Immunoassay; Biospecific binding assay for micro-organisms, e.g. protozoa, bacteria, viruses
-
- G—PHYSICS
- G01—MEASURING; TESTING
- G01N—INVESTIGATING OR ANALYSING MATERIALS BY DETERMINING THEIR CHEMICAL OR PHYSICAL PROPERTIES
- G01N33/00—Investigating or analysing materials by specific methods not covered by the preceding groups
- G01N33/48—Investigating or analysing materials by specific methods not covered by the preceding groups biological material, e.g. blood, urine; Haemocytometers
- G01N33/50—Chemical analysis of biological material, e.g. blood, urine; Testing involving biospecific ligand binding methods; Immunological testing
- G01N33/53—Immunoassay; Biospecific binding assay
- G01N33/564—Immunoassay; Biospecific binding assay for pre-existing immune complex or autoimmune disease, i.e. systemic lupus erythematosus, rheumatoid arthritis, multiple sclerosis, rheumatoid factors or complement components C1-C9
-
- G—PHYSICS
- G01—MEASURING; TESTING
- G01N—INVESTIGATING OR ANALYSING MATERIALS BY DETERMINING THEIR CHEMICAL OR PHYSICAL PROPERTIES
- G01N33/00—Investigating or analysing materials by specific methods not covered by the preceding groups
- G01N33/48—Investigating or analysing materials by specific methods not covered by the preceding groups biological material, e.g. blood, urine; Haemocytometers
- G01N33/50—Chemical analysis of biological material, e.g. blood, urine; Testing involving biospecific ligand binding methods; Immunological testing
- G01N33/5005—Chemical analysis of biological material, e.g. blood, urine; Testing involving biospecific ligand binding methods; Immunological testing involving human or animal cells
- G01N33/5008—Chemical analysis of biological material, e.g. blood, urine; Testing involving biospecific ligand binding methods; Immunological testing involving human or animal cells for testing or evaluating the effect of chemical or biological compounds, e.g. drugs, cosmetics
- G01N33/5044—Chemical analysis of biological material, e.g. blood, urine; Testing involving biospecific ligand binding methods; Immunological testing involving human or animal cells for testing or evaluating the effect of chemical or biological compounds, e.g. drugs, cosmetics involving specific cell types
- G01N33/5047—Cells of the immune system
-
- G—PHYSICS
- G01—MEASURING; TESTING
- G01N—INVESTIGATING OR ANALYSING MATERIALS BY DETERMINING THEIR CHEMICAL OR PHYSICAL PROPERTIES
- G01N33/00—Investigating or analysing materials by specific methods not covered by the preceding groups
- G01N33/48—Investigating or analysing materials by specific methods not covered by the preceding groups biological material, e.g. blood, urine; Haemocytometers
- G01N33/50—Chemical analysis of biological material, e.g. blood, urine; Testing involving biospecific ligand binding methods; Immunological testing
- G01N33/80—Chemical analysis of biological material, e.g. blood, urine; Testing involving biospecific ligand binding methods; Immunological testing involving blood groups or blood types or red blood cells
-
- G—PHYSICS
- G01—MEASURING; TESTING
- G01N—INVESTIGATING OR ANALYSING MATERIALS BY DETERMINING THEIR CHEMICAL OR PHYSICAL PROPERTIES
- G01N33/00—Investigating or analysing materials by specific methods not covered by the preceding groups
- G01N33/48—Investigating or analysing materials by specific methods not covered by the preceding groups biological material, e.g. blood, urine; Haemocytometers
- G01N33/50—Chemical analysis of biological material, e.g. blood, urine; Testing involving biospecific ligand binding methods; Immunological testing
- G01N33/68—Chemical analysis of biological material, e.g. blood, urine; Testing involving biospecific ligand binding methods; Immunological testing involving proteins, peptides or amino acids
-
- G—PHYSICS
- G01—MEASURING; TESTING
- G01N—INVESTIGATING OR ANALYSING MATERIALS BY DETERMINING THEIR CHEMICAL OR PHYSICAL PROPERTIES
- G01N33/00—Investigating or analysing materials by specific methods not covered by the preceding groups
- G01N33/48—Investigating or analysing materials by specific methods not covered by the preceding groups biological material, e.g. blood, urine; Haemocytometers
- G01N33/483—Physical analysis of biological material
- G01N33/487—Physical analysis of biological material of liquid biological material
-
- G—PHYSICS
- G01—MEASURING; TESTING
- G01N—INVESTIGATING OR ANALYSING MATERIALS BY DETERMINING THEIR CHEMICAL OR PHYSICAL PROPERTIES
- G01N2800/00—Detection or diagnosis of diseases
Similar Documents
| Publication | Publication Date | Title |
|---|---|---|
| Koc et al. | Toll-like receptor expression in monocytes in patients with chronic kidney disease and haemodialysis: relation with inflammation | |
| Karsten et al. | Red blood cells are dynamic reservoirs of cytokines | |
| Hu et al. | Discovering the secret of diseases by incorporated tear exosomes analysis via rapid-isolation system: iTEARS | |
| Belmouaz et al. | Comparison of the removal of uraemic toxins with medium cut-off and high-flux dialysers: a randomized clinical trial | |
| Kwon et al. | Successful pre-transplant management of a patient with anti-factor H autoantibodies-associated haemolytic uraemic syndrome | |
| Eskandary et al. | ABO antibody and complement depletion by immunoadsorption combined with membrane filtration—a randomized, controlled, cross-over trial | |
| Sester et al. | Strong depletion of CD14+ CD16+ monocytes during haemodialysis treatment | |
| Ando et al. | Impairment of innate cellular response to in vitro stimuli in patients on continuous ambulatory peritoneal dialysis | |
| Grabulosa et al. | Chronic kidney disease induces inflammation by increasing Toll-like receptor-4, cytokine and cathelicidin expression in neutrophils and monocytes | |
| Kneis et al. | Elimination of middle-sized uremic solutes with high-flux and high-cut-off membranes: a randomized in vivo study | |
| Herget‐Rosenthal et al. | Modulation and source of procalcitonin in reduced renal function and renal replacement therapy | |
| Merino et al. | Losartan prevents the development of the pro-inflammatory monocytes CD14+ CD16+ in haemodialysis patients | |
| Liakopoulos et al. | Hemodialysis-related changes in phenotypical features of monocytes | |
| Milan Manani et al. | Pro-inflammatory cytokines: a possible relationship with dialytic adequacy and serum albumin in peritoneal dialysis patients | |
| Zhu et al. | Correlation of increased Th17/Treg cell ratio with endoplasmic reticulum stress in chronic kidney disease | |
| Saad et al. | Lymphocyte populations and apoptosis of peripheral blood B and T lymphocytes in children with end stage renal disease | |
| van Tellingen et al. | Enhanced long-term reduction of plasma leptin concentrations by super-flux polysulfone dialysers | |
| Ankersmit et al. | Impaired T cell proliferation, increased soluble death-inducing receptors and activation-induced T cell death in patients undergoing haemodialysis | |
| Musiał et al. | Serum VCAM-1, ICAM-1, and L-selectin levels in children and young adults with chronic renal failure | |
| Eberhardson et al. | Randomised, double-blind, placebo-controlled trial of CCR9-targeted leukapheresis treatment of ulcerative colitis patients | |
| Hesselink et al. | The effects of chronic kidney disease and renal replacement therapy on circulating dendritic cells | |
| Vaslaki et al. | On‐line hemodiafiltration does not induce inflammatory response in end‐stage renal disease patients: results from a multicenter cross‐over study | |
| Kakuta et al. | A Prospective Multicenter Randomized Controlled Study on Interleukin‐6 Removal and Induction by a new Hemodialyzer With Improved Biocompatibility in Hemodialysis Patients: A Pilot Study | |
| Cole et al. | The effect of coupled haemofiltration and adsorption on inflammatory cytokines in an ex vivo model | |
| Ito et al. | Neutrophil/lymphocyte ratio elevation in renal dysfunction is caused by distortion of leukocyte hematopoiesis in bone marrow |